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Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

June 19, 2020COVID-19, Vizit Graphs
Possibly implicated genes in SARS-CoV-2-mediated blood vessel diseaseEftychia Lekka2020-08-03T14:07:20-04:00
Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

In addition to respiratory symptoms, novel findings have shown vascular complications in a series of patients with COVID-19, suggesting that COVID-19 is more to a respiratory infection caused by SARS-CoV-2. This graph illustrates some of the possible mechanisms (genes) by which these effects of SARS-CoV-2 may be mediated.

Explore the live graph.

Tags: ACE, ACE2, ALBUMIN, ANGIOTENSIN II, APOE, BLOOD VESSEL DISEASE, COVID-19, FIBRINOGEN, HEMOGLOBIN, ICAM1, IL1B, IL6, LACTATE DEHYDROGENASE, NFKB1, RAS, SARS-COV-2, THROMBIN, TNF, VEGFA

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← Adverse Event considerations for repositioning remdesivir, hydroxychloroquine, chloroquine, and interleukin (IL)-6 inhibitors in COVID-19 patients
ACE2, TMPRSS2, Acute Lung Injury, ARDS, and Lung Fibrosis →
VASODILATION Uberon TYPE 2 DIABETES TUMOR PROGRESSION THORAX VIRUS DISEASES VASCULAR TAU TREPONEMA DENTICOLA VASOCONSTRICTION TAUOPATHIES TRASTUZUMAB TUMOR TECFIDERA TRACHEA TROGLITAZONE VEGF TISSUE DEVELOPMENT TRANSLATION UBIQUITINATION VITAMIN D THROMBOCYTOPENIA TARGET LESION IDENTIFICATION X-RAY COMPUTED TOMOGRAPHY THROMBOSIS VGF PROTEIN TMPRSS2 TIMELESS TRANSPORT VEGFA TGF-BETA THY-1 ANTIGENS TH17 CELLS TROPISM TP53 ULCERATIVE COLITIS VEINS VERO CELLS UCHL1 PROTEIN VIRAL RELEASE FROM HOST CELL WOUND HEALING TUMOR GROWTH THROMBIN

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