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Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

June 19, 2020COVID-19, Vizit Graphs
Possibly implicated genes in SARS-CoV-2-mediated blood vessel diseaseEftychia Lekka2020-08-03T14:07:20-04:00
Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

In addition to respiratory symptoms, novel findings have shown vascular complications in a series of patients with COVID-19, suggesting that COVID-19 is more to a respiratory infection caused by SARS-CoV-2. This graph illustrates some of the possible mechanisms (genes) by which these effects of SARS-CoV-2 may be mediated.

Explore the live graph.

Tags: ACE, ACE2, ALBUMIN, ANGIOTENSIN II, APOE, BLOOD VESSEL DISEASE, COVID-19, FIBRINOGEN, HEMOGLOBIN, ICAM1, IL1B, IL6, LACTATE DEHYDROGENASE, NFKB1, RAS, SARS-COV-2, THROMBIN, TNF, VEGFA

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← Dexamethasone mechanism of action analysis in the treatment of SARS-COV-2 induced infections
ACE2, TMPRSS2, Acute Lung Injury, ARDS, and Lung Fibrosis →
TUMOR PROGRESSION THY-1 ANTIGENS TH17 CELLS TROPISM UCHL1 PROTEIN TYPE 2 DIABETES TRANSLATION TMPRSS2 TISSUE DEVELOPMENT TUMOR TAUOPATHIES VERO CELLS VEGFA UBIQUITINATION VASOCONSTRICTION VITAMIN D VIRUS DISEASES X-RAY COMPUTED TOMOGRAPHY TREPONEMA DENTICOLA TRACHEA TECFIDERA VIRAL RELEASE FROM HOST CELL TRANSPORT VEINS TIMELESS VGF PROTEIN Uberon TP53 THROMBOCYTOPENIA TRASTUZUMAB TARGET LESION IDENTIFICATION THROMBOSIS ULCERATIVE COLITIS THROMBIN TGF-BETA WOUND HEALING TAU VEGF TUMOR GROWTH VASODILATION TROGLITAZONE VASCULAR THORAX

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