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Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

June 19, 2020COVID-19, Vizit Graphs
Possibly implicated genes in SARS-CoV-2-mediated blood vessel diseaseEftychia Lekka2020-08-03T14:07:20-04:00
Possibly implicated genes in SARS-CoV-2-mediated blood vessel disease

In addition to respiratory symptoms, novel findings have shown vascular complications in a series of patients with COVID-19, suggesting that COVID-19 is more to a respiratory infection caused by SARS-CoV-2. This graph illustrates some of the possible mechanisms (genes) by which these effects of SARS-CoV-2 may be mediated.

Explore the live graph.

Tags: ACE, ACE2, ALBUMIN, ANGIOTENSIN II, APOE, BLOOD VESSEL DISEASE, COVID-19, FIBRINOGEN, HEMOGLOBIN, ICAM1, IL1B, IL6, LACTATE DEHYDROGENASE, NFKB1, RAS, SARS-COV-2, THROMBIN, TNF, VEGFA

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← Dexamethasone mechanism of action analysis in the treatment of SARS-COV-2 induced infections
ACE2, TMPRSS2, Acute Lung Injury, ARDS, and Lung Fibrosis →
TARGET LESION IDENTIFICATION TRANSLATION TAUOPATHIES TIMELESS UCHL1 PROTEIN ULCERATIVE COLITIS VEGF TP53 TISSUE DEVELOPMENT VASODILATION TECFIDERA TUMOR GROWTH VASOCONSTRICTION THROMBIN TREPONEMA DENTICOLA THROMBOSIS VIRUS DISEASES X-RAY COMPUTED TOMOGRAPHY THORAX THY-1 ANTIGENS TAU TROPISM Uberon TRANSPORT TMPRSS2 TYPE 2 DIABETES VITAMIN D TROGLITAZONE TUMOR PROGRESSION VGF PROTEIN WOUND HEALING TH17 CELLS VIRAL RELEASE FROM HOST CELL VEGFA VEINS TUMOR TGF-BETA TRACHEA THROMBOCYTOPENIA UBIQUITINATION TRASTUZUMAB VERO CELLS VASCULAR

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